Online Purchasing Account You are logged on as Guest. LoginRegister a New AccountShopping cart (Empty)
United States 

Hinokitiol

Chelator that can induce differentiation in tumor cells
 
BML-FR110-0250 250 mg 219.00 USD
Do you need bulk/larger quantities?
 
Hinokitiol (β-thujaplicin) is a potent metal chelator that induces differentiation and apoptosis in teratocarcinoma F9 cells and tyrosinase, most likely via metal chelation.

Product Specification

MW:164.2
 
CAS:499-44-5
 
Purity:≥98% (GC)
 
Shipping:Ambient
 
Long Term Storage:Ambient
 
Regulatory Status:RUO - Research Use Only
 
bml-fr110
Please mouse over
bml-fr110

Product Literature References

(-)-Epigallocatechin-3-gallate and hinokitiol reduce melanin synthesis via decreased MITF production: D. S. Kim, et al.; Arch. Pharm. Res. 27, 334 (2004), Abstract;
Hinokitiol, a selective inhibitor of the platelet-type isozyme of arachidonate 12-lipoxygenase: H. Suzuki, et al.; Biochem. Biophys. Res. Commun. 275, 885 (2000), Abstract;
Antimicrobial activity and metalloprotease inhibition of hinokitiol-related compounds, the constituents of Thujopsis dolabrata S. and Z. hondai MAK: Y. Inamori, et al.; Biol. Pharm. Bull. 22, 990 (1999), Abstract;
Apoptosis during iron chelator-induced differentiation in F9 embryonal carcinoma cells: T. Tanaka, et al.; Cell Biol. Int. 23, 541 (1999), Abstract;
Induction of apoptosis by hinokitiol, a potent iron chelator, in teratocarcinoma F9 cells is mediated through the activation of caspase-3: Y. Ido, et al.; Cell Prolif. 32, 63 (1999), Abstract;
Inhibitory effect of beta-thujaplicin on ultraviolet B-induced apoptosis in mouse keratinocytes: T. Baba, et al.; J. Invest. Dermatol. 110, 24 (1998), Abstract; Full Text

Related Literature

Brochures
Comprehensive Tools for Quantifying Cellular Responses to Oxidative Damage
Comprehensive Tools for Quantifying Cellular Responses to Oxidative Damage
Download as PDF

All new literature pieces

Recommend this page

 
Keep in touch

©2020 Enzo Life Sciences, Inc.,