Product Details
| Alternative Name: | BCL2L4, BCL2-associated X protein |
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| Host: | Rabbit |
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| Immunogen: | Synthetic peptide corresponding to aa 43-61 (P43ELTLEQPPQDASTKKLSE61) of mouse Bax. This sequence is completely conserved in rat Bax. |
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| UniProt ID: | Q07813 |
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| Species reactivity: | Human, Mouse, Rat
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| Applications: | IHC (FS), IHC (PS), IP, WB
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| Recommended Dilutions/Conditions: | Immunohistochemistry (paraffin sections, 1:500-1:2,000)
Immunoprecipitation (1µl/106 cells)
Western Blot (1:500-1:2,000)
Suggested dilutions/conditions may not be available for all applications.
Optimal conditions must be determined individually for each application. |
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| Formulation: | Liquid. In PBS containing 0.05% sodium azide. |
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| Use/Stability: | Stable for at least 1 year after receipt when stored at -80°C. |
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| Shipping: | Blue Ice |
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| Short Term Storage: | +4°C |
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| Long Term Storage: | -80°C |
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| Regulatory Status: | RUO - Research Use Only |
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Product Literature References
The gastroprotective effects of hydroalcoholic extract of Monolluma quadrangula against ethanol-induced gastric mucosal injuries in Sprague Dawley rats: I.A.A. Ibrahim, et al.; Drug Des. Devel. Ther.
10, 93 (2016),
Application(s): Western blot,
Abstract;
Full Text
Mcl-1 is a key regulator of the ovarian reserve: S. Omari, et al.; Cell. Death Dis.
6, e1755 (2015),
Application(s): Immunofluorescence,
Abstract;
Full Text
Sequential cleavage of poly(ADP-ribose)polymerase and appearance of a small Bax-immunoreactive protein are blocked by Bcl-X(L) and caspase inhibitors during staurosporine-induced dopaminergic neuronal apoptosis: J.E. Kim, et al.; J. Neurochem.
72, 2456 (1999),
Abstract;
Full Text
Bax cleavage is mediated by calpain during drug-induced apoptosis: D.E. Wood, et al.; Oncogene
17, 1069 (1998),
Abstract;
Developmental expression patterns of Bcl-2, Bcl-x, Bax, and Bak in teeth: S. Krajewski, et al.; Cell Death Differ.
5, 408 (1998),
Abstract;
Apoptosis in the terminal endbud of the murine mammary gland: a mechanism of ductal morphogenesis: R.C. Humphreys, et al.; Development
122, 4013 (1996),
Abstract;
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Expression of Bcl-2, Bcl-x, and Bax after T cell activation and IL-2 withdrawal: H.E. Broome, et al.; J. Immunol.
155, 2311 (1995),
Abstract;
Overexpression of the Bcl-2 protein increases the half-life of p21Bax: T. Miyashita, et al.; J. Biol. Chem.
270, 26049 (1995),
Abstract;
Full Text
Upregulation of bax protein levels in neurons following cerebral ischemia: S. Krajewski, et al.; J. Neurosci.
15, 6364 (1995),
Abstract;
Immunohistochemical determination of in vivo distribution of Bax, a dominant inhibitor of Bcl-2: S. Krajewski, et al.; Am. J. Pathol.
145, 1323 (1994),
Abstract;
