Online Purchasing Account You are logged on as Guest. LoginRegister a New AccountShopping cart (Empty)
United States 


Chelator that can induce differentiation in tumor cells
BML-FR110-0250 250 mg 213.00 USD
Do you need bulk/larger quantities?
Hinokitiol (β-thujaplicin) is a potent metal chelator that induces differentiation and apoptosis in teratocarcinoma F9 cells and tyrosinase, most likely via metal chelation.

Product Specification

Purity:≥98% (GC)
Long Term Storage:Ambient
Please mouse over

Product Literature References

(-)-Epigallocatechin-3-gallate and hinokitiol reduce melanin synthesis via decreased MITF production: D. S. Kim, et al.; Arch. Pharm. Res. 27, 334 (2004), Abstract;
Hinokitiol, a selective inhibitor of the platelet-type isozyme of arachidonate 12-lipoxygenase: H. Suzuki, et al.; Biochem. Biophys. Res. Commun. 275, 885 (2000), Abstract;
Antimicrobial activity and metalloprotease inhibition of hinokitiol-related compounds, the constituents of Thujopsis dolabrata S. and Z. hondai MAK: Y. Inamori, et al.; Biol. Pharm. Bull. 22, 990 (1999), Abstract;
Apoptosis during iron chelator-induced differentiation in F9 embryonal carcinoma cells: T. Tanaka, et al.; Cell Biol. Int. 23, 541 (1999), Abstract;
Induction of apoptosis by hinokitiol, a potent iron chelator, in teratocarcinoma F9 cells is mediated through the activation of caspase-3: Y. Ido, et al.; Cell Prolif. 32, 63 (1999), Abstract;
Inhibitory effect of beta-thujaplicin on ultraviolet B-induced apoptosis in mouse keratinocytes: T. Baba, et al.; J. Invest. Dermatol. 110, 24 (1998), Abstract; Full Text

Related Literature

Comprehensive Tools for Quantifying Cellular Responses to Oxidative Damage
Comprehensive Tools for Quantifying Cellular Responses to Oxidative Damage
Download as PDF

Technical Posters
Disease-Associated Stress Signaling
Disease-Associated Stress Signaling
Download as PDF

All new literature pieces

Recommend this page

For Research Use Only. Not for use in diagnostic procedures.
Keep in touch

©2018 Enzo Life Sciences, Inc.,