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Cytochalasin E

Inhibits angiogenesis
 
BML-CT120-0005 5 mg 223.00 USD
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Replaces Prod. #: ALX-380-062

An aspergillus derived fungal metabolite. Inhibits angiogenesis and tumor growth. An actin microfilament depolymerizing agent.

Product Specification

Formula:C28H33NO7
 
MW:495.6
 
Source:Isolated from Aspergillus clavatus
 
CAS:36011-19-5
 
Purity:≥98% (HPLC, TLC)
 
Appearance:White solid.
 
Solubility:Soluble in DMSO (50mg/ml) or ethyl acetate (5mg/ml)
 
Shipping:Ambient
 
Long Term Storage:-20°C
 
Use/Stability:Store, as supplied, at -20°C for up to 1 year. Store solutions at -20°C for up to 3 months.
 
Handling:Protect from light.
 
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Product Literature References

Depolymerization of actin filament by cytochalasin E induces interleukin-8 production and up-regulates CD54 in the HeLa epithelial cell line: N. Ikewai et al.; Microbiol. Immunol. 47, 775 (2003), Abstract;
Actin depolymerization and polymerization are required during apoptosis in endothelial cells: N. Suarez-Huerta et al.; J. Cell. Physiol. 184, 239 (2000), Abstract;
Cytochalasin E, an epoxide containing Aspergillus-derived fungal metabolite, inhibits angiogenesis and tumor growth: T. Udagawa et al.; J. Pharmacol. Exp. Ther. 294, 421 (2000), Abstract;
Cytochalasin-E-Resistant Mutants of Coprinus cinereus: Isolation and Genetic, Biochemical, and Cytological Analyses: M. Tsukamoto et al.; Fungal Genet. Biol. 20, 52 (1996), Abstract;
Inhibition of actin polymerization in blood platelets by cytochalasins: J.E. Fox & D.R. Phillips; Nature 292, 650 (1981), Abstract;

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